Pathological Effects of Trypanosoma Brucei on Small Blood Vessels in Rabbit Ear-Chambers

dc.bibliographicCitation.endpage88en
dc.bibliographicCitation.issue1en
dc.bibliographicCitation.stpage82en
dc.bibliographicCitation.titleTransactions of The Royal Society of Tropical Medicine And Hygieneen
dc.bibliographicCitation.volume65en
dc.contributor.authorGOODWIN, L. G.null
dc.contributor.institutionNuffield Institute of Comparative Medicine, The Zoological Society of London, Regent's Park, London, N.W.1 UK
dc.date.accessioned2015-08-24T11:12:29Znull
dc.date.available2015-08-24T11:12:29Znull
dc.date.issued1971en
dc.description.abstractInfection with trypanosomes of the Trypanosoma brucei subgroup causes the liberation of short-chain peptides with kinin-like activity in mice, rats, rabbits and cattle (GOODWIN and RICHARDS, 1960; RICHARDS, 1965; BOREHAM, 1966, 1968) and also in man (BOREHAM and GOODWIN, 1969). The release of kinin is associated with antigen: antibody reactions and may be responsible for changes in vascular permeability that lead to the production of oedema. A study of the blood vessels of the ears and the cremaster muscles of rabbits showed that infection with T. brucei led to a chronic vasculitis (GOODWIN and HOOK, 1968).en
dc.description.sponsorshipMinistry of overseas Development
dc.identifier.citationGoodwin, L. G. (1971). Pathological effects of Trypanosoma brucei on small blood vessels in rabbit ear-chambers. Transactions of the Royal Society of Tropical Medicine and Hygiene, 65(1), 82-88.https://doi.org/10.1016/0035-9203(71)90189-1en
dc.identifier.doihttps://doi.org/10.1016/0035-9203(71)90189-1
dc.identifier.issn0035-9203*
dc.identifier.urihttp://kalroerepository.kalro/org/handle/0/11003null
dc.language.isoenglishen
dc.rights.urihttp://creativecommons.org/licenses/by/3.0/en
dc.subject.agrovocTrypanosoma bruceien
dc.subject.agrovocEarsen
dc.subject.agrovocBlood vesselsen
dc.subject.agrovocPathologyen
dc.titlePathological Effects of Trypanosoma Brucei on Small Blood Vessels in Rabbit Ear-Chambersen
dc.typeJournal Contribution*
dc.type.refereedRefereeden
dc.type.specifiedArticleen

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